The
National Veterinary Research Institute has announced that it is looking
into developing a method of testing for African Swine Fever (ASF),
while a ‘biosecurity’ project is also initiated.
http://www.thenews.pl/1/12/Artykul/205136,New-method-for-detecting-African-Swine-Fever-being-developed
Wednesday, April 29, 2015
Wednesday, April 22, 2015
UK Pig farmers call for wild boar cull
http://www.fwi.co.uk/news/pig-farmers-call-for-wild-boar-cull.htm
"African swine fever would burn itself out if it was only in the wild boar population. But the disease was persisting because it was jumping in and out of the backyard pig population."
"African swine fever would burn itself out if it was only in the wild boar population. But the disease was persisting because it was jumping in and out of the backyard pig population."
Monday, April 20, 2015
Similarities Between Pig Virus and AIDS Explored in ABC News Commentary
http://kaiserhealthnews.org/morning-breakout/dr00003772/
"Regush writes that PRRS and AIDS share several similar traits."
"Regush writes that PRRS and AIDS share several similar traits."
Friday, April 17, 2015
Have they looked for an underlying infection of a brand new strain of African Swine Fever Virus or HHV-6, HHV-7, HHV-8, PLHV-1 or PLHV-2?
Fatal disease associated with Swine Hepatitis E virus and Porcine circovirus 2 co-infection in four weaned pigs in China
https://www.pig333.com/swine_abstracts/fatal-disease-associated-with-swine-hev-and-pcv2_9973/#.VTD-XiENBH8.twitter
Thursday, April 16, 2015
Modulatory effects on dendritic cells by human herpesvirus 6 and African Swine Fever Virus
http://journal.frontiersin.org/article/10.3389/fmicb.2015.00388/abstract
"Human herpesvirus 6A and 6B are β-herpesviruses approaching 100% seroprevalance worldwide. These viruses are involved in several clinical syndromes and have important immunomodulatory effects. Dendritic cells (DC) are key players in innate and adaptive immunity. Accordingly, DC are implicated in the pathogenesis of many human diseases, including infections. In this review the effects of HHV-6 infection on DC will be discussed."
Wikipedia on Dendritic Cells:
Dendritic cells (DCs) are antigen-presenting cells (also known as accessory cells) of the mammalian immune system. Their main function is to process antigen material and present it on the cell surface to the T cells of the immune system. They act as messengers between the innate and the adaptive immune systems.
Dendritic cells are present in those tissues that are in contact with the external environment, such as the skin (where there is a specialized dendritic cell type called the Langerhans cell) and the inner lining of the nose, lungs, stomach and intestines. They can also be found in an immature state in the blood. Once activated, they migrate to the lymph nodes where they interact with T cells and B cells to initiate and shape the adaptive immune response. At certain development stages they grow branched projections, the dendrites that give the cell its name (δένδρον or déndron being Greek for "tree"). While similar in appearance, these are structures distinct from the dendrites of neurons. Immature dendritic cells are also called veiled cells, as they possess large cytoplasmic 'veils' rather than dendrites.
To compare HHV-6 and African Swine Fever's affect on dendritic cells, click here.
"Human herpesvirus 6A and 6B are β-herpesviruses approaching 100% seroprevalance worldwide. These viruses are involved in several clinical syndromes and have important immunomodulatory effects. Dendritic cells (DC) are key players in innate and adaptive immunity. Accordingly, DC are implicated in the pathogenesis of many human diseases, including infections. In this review the effects of HHV-6 infection on DC will be discussed."
Wikipedia on Dendritic Cells:
Dendritic cells (DCs) are antigen-presenting cells (also known as accessory cells) of the mammalian immune system. Their main function is to process antigen material and present it on the cell surface to the T cells of the immune system. They act as messengers between the innate and the adaptive immune systems.
Dendritic cells are present in those tissues that are in contact with the external environment, such as the skin (where there is a specialized dendritic cell type called the Langerhans cell) and the inner lining of the nose, lungs, stomach and intestines. They can also be found in an immature state in the blood. Once activated, they migrate to the lymph nodes where they interact with T cells and B cells to initiate and shape the adaptive immune response. At certain development stages they grow branched projections, the dendrites that give the cell its name (δένδρον or déndron being Greek for "tree"). While similar in appearance, these are structures distinct from the dendrites of neurons. Immature dendritic cells are also called veiled cells, as they possess large cytoplasmic 'veils' rather than dendrites.
To compare HHV-6 and African Swine Fever's affect on dendritic cells, click here.
The Joint Research Institute that Russian and European Scientists Need to Start Immediately
This should be headquartered in Paris, Madrid, Stockholm, Rome, London or Moscow:
The
Russian and European Institute for the study of the Interaction of
HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Infections
in Humans and their Implications for Human Health and Pork Consumption.
Eight Divisions of the Russian and European Institute:
1. The Division for the study of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus in Autism Spectrum Disorders.
4. The Division for the Study of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus in Multiple Sclerosis.
5. The Division for the study of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus in emerging cancer epidemics in humans and pigs.
6. The Division for the study of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus in emerging autoimmune disorder epidemics in humans and pigs.
7. The Division for the study of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus in emerging neurologic/psychologic disorders.
8. The Division for the study of the control of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus on Farms.
9. The Division for the study of the impact of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus on the health of farm workers and people who visit farms.
10. The Division for the study of the impact of HHV-6, HHV-7, HHV-8, PLHV-1, PLHV-2 and African Swine Fever Virus on humans who handle or consume pork infected with these viruses.
Wednesday, April 15, 2015
The Big HHV-6/7/8 Experiment Russian And European Scientist Need to Conduct Immediately
Special Notice to Russian and European Scientists:
If you want to really understand the human threat of African Swine Fever in Russia and Europe, conduct the following experiment.
1. Infect three sets of disease-free pigs with HHV-6, HHV-7 and HHV-8. (Use controls, of course.)
2. Monitor the health of the pigs.
3. Determine if the HHV-6, HHV-7, and HHV-8 infected pigs develop symptoms consistent with autism, chronic fatigue syndrome or AIDS or a spectrum or all three.
4. Monitor the changes in the immune systems of all the pigs.
5. Determine if the HHV-6, HHV-7 and HHV-8 infected pigs develop opportunistic infections consistent with immune dysfunction.
6. Examine the pigs for any signs of hemorrhagic lesions resembling Kaposi's sarcoma in humans.
7. Test the pigs for a broad spectrum of antibodies to African Swine Fever.
8. Isolate the HHV-6, HHV-7 and HHV-8 from the infected pigs and compare the viruses to African Swine Fever Virus.
9. Try to isolate viruses that resemble African Swine Fever from the HHV-6, HHV-7 and HHV-8 infected pigs.
This will help determine if African Swine Fever is really a kind of HHV-6/7/8 spectrum disease, and whether HHV-6/7/8 in humans is a kind of African Swine Fever spectrum disease in humans.
Consider testing all pork products for HHV-6, HHV-7 and HHV-8.
Consider screening the human populations of Russia and Europe for all strains of African Swine Fever Virus.
For more information about the potentially intertwined nature of the epidemics of HHV-8 and African Swine Fever Virus in Sardinia click here.
For more information on a vaccine against African Swine Fever that may help protect humans click here.
To learn more about John Beldekas, scientist who accused discredited American scientist Robert Gallo of appropriating his work on ASFV and giving African Swine Fever the new name of HHV-6, click here (and scroll down to Beldekas story).
To learn more about all the crimes committed in the laboratory of Robert Gallo, click here and read this book by John Crewdson.
If you want to really understand the human threat of African Swine Fever in Russia and Europe, conduct the following experiment.
1. Infect three sets of disease-free pigs with HHV-6, HHV-7 and HHV-8. (Use controls, of course.)
2. Monitor the health of the pigs.
3. Determine if the HHV-6, HHV-7, and HHV-8 infected pigs develop symptoms consistent with autism, chronic fatigue syndrome or AIDS or a spectrum or all three.
4. Monitor the changes in the immune systems of all the pigs.
5. Determine if the HHV-6, HHV-7 and HHV-8 infected pigs develop opportunistic infections consistent with immune dysfunction.
6. Examine the pigs for any signs of hemorrhagic lesions resembling Kaposi's sarcoma in humans.
7. Test the pigs for a broad spectrum of antibodies to African Swine Fever.
8. Isolate the HHV-6, HHV-7 and HHV-8 from the infected pigs and compare the viruses to African Swine Fever Virus.
9. Try to isolate viruses that resemble African Swine Fever from the HHV-6, HHV-7 and HHV-8 infected pigs.
This will help determine if African Swine Fever is really a kind of HHV-6/7/8 spectrum disease, and whether HHV-6/7/8 in humans is a kind of African Swine Fever spectrum disease in humans.
Consider testing all pork products for HHV-6, HHV-7 and HHV-8.
Consider screening the human populations of Russia and Europe for all strains of African Swine Fever Virus.
For more information about the potentially intertwined nature of the epidemics of HHV-8 and African Swine Fever Virus in Sardinia click here.
For more information on a vaccine against African Swine Fever that may help protect humans click here.
To learn more about John Beldekas, scientist who accused discredited American scientist Robert Gallo of appropriating his work on ASFV and giving African Swine Fever the new name of HHV-6, click here (and scroll down to Beldekas story).
To learn more about all the crimes committed in the laboratory of Robert Gallo, click here and read this book by John Crewdson.
Tuesday, April 14, 2015
The Big Question for Russian and European Scientists about PRRS and African Swine Fever Virus
Serious Russian and European scientists interested in African Swine Fever Virus research should ask these questions:
PRRS hit America at the time that African Swine Fever was expected to break out. Why didn't ASFV break out in the United States in the late 70s and early 80s? Maybe it did. Did scientists either intentionally or unintentionally rename ASFV, calling it PRRS?
Is the inability to control PRRS due to the fact that it really is a new form of ASFV which is causing all kinds of opportunistic infections in pigs?
Is what is thought to be the PRRS virus just an opportunistic problem of an underlying infection by a new strain of African Swine Fever virus?
And is it possible that all of this confusion (deliberate or inadvertent) has affected human health?
Is testing for new strains of African Swine Fever Virus a total diagnostic mess?
Is it safer for countries with ASFV outbreaks to not test for ASFV in pigs (or people) and pretend that they did?
https://www.pig333.com/prrs/prrsv-in-europe-where-did-it-come-from_9894/
PRRS hit America at the time that African Swine Fever was expected to break out. Why didn't ASFV break out in the United States in the late 70s and early 80s? Maybe it did. Did scientists either intentionally or unintentionally rename ASFV, calling it PRRS?
Is the inability to control PRRS due to the fact that it really is a new form of ASFV which is causing all kinds of opportunistic infections in pigs?
Is what is thought to be the PRRS virus just an opportunistic problem of an underlying infection by a new strain of African Swine Fever virus?
And is it possible that all of this confusion (deliberate or inadvertent) has affected human health?
Is testing for new strains of African Swine Fever Virus a total diagnostic mess?
Is it safer for countries with ASFV outbreaks to not test for ASFV in pigs (or people) and pretend that they did?
PRRSV in Europe: where did it come from?
https://www.pig333.com/prrs/prrsv-in-europe-where-did-it-come-from_9894/
28% of Farmers in Madagascar believe African Swine Fever Virus Can Affect Humans
"Believing that ASF can affect humans, the farmers sell the meat although
that is illegal in the country, according to a new study of farmers'
behaviour."
"Almost three-quarters of farmers questioned said they have sold meat from pigs infected with African swine fever (ASF) despite the fact that the government has outlawed this practice."
http://www.thepigsite.com/swinenews/39123/lawbreakers-add-to-african-swine-fever-challenges-in-madagascar/
"Almost three-quarters of farmers questioned said they have sold meat from pigs infected with African swine fever (ASF) despite the fact that the government has outlawed this practice."
http://www.thepigsite.com/swinenews/39123/lawbreakers-add-to-african-swine-fever-challenges-in-madagascar/
Viruses Russian and European scientists should look for in sera from American and British pigs:
1. HHV-6
2. HHV-7
3. HHV-8
4. Porcine Lymphotropic Herpesvirus 1
5. Porcine Lymphotropic Herpesvirus 2
6. African Swine Fever Virus
2. HHV-7
3. HHV-8
4. Porcine Lymphotropic Herpesvirus 1
5. Porcine Lymphotropic Herpesvirus 2
6. African Swine Fever Virus
Viruses European and Russian researchers should look for in American and British pork products:
1. HHV-6
2. HHV-7
3. HHV-8
4. Porcine Lymphotropic Herpesvirus 1
5. Porcine Lymphotropic Herpesvirus 2
6. African Swine Fever Virus
2. HHV-7
3. HHV-8
4. Porcine Lymphotropic Herpesvirus 1
5. Porcine Lymphotropic Herpesvirus 2
6. African Swine Fever Virus
Monday, April 13, 2015
Are American AIDS Researchers Geniuses or Crooks and Charlatans?
The one book every Russian and European scientist should read immediately!
http://www.amazon.com/Science-Sold-Out-Really-Cause/dp/1556436424
Sunday, April 12, 2015
Pigs, Kaposi's Sarcoma and African Swine Fever: Is HHV-8 the human infection of ASFV in Sardinia?
Special alert to Russian and European scientists:
http://www.ncbi.nlm.nih.gov/pubmed/9607855
High prevalence of antibodies to human herpesvirus 8 in relatives of patients with classic Kaposi's sarcoma from Sardinia.
http://www.ncbi.nlm.nih.gov/pubmed/9607855
Infection with human herpesvirus type 8 and Kaposi's sarcoma in Sardinia.
http://www.ncbi.nlm.nih.gov/pubmed/16501902
Epidemiology of HHV8 in Sardinian emigrants
http://www.dsnm.univr.it/?ent=progetto&id=565
Both ASFV and HHV-8 Interfere with apoptosis.
Are ASFV-infected Pigs the viral source of HHV-8 related Kaposi's Sarcoma in Sardinia? Is a ASFV-related Kaposi's Sarcoma epidemic possible in Russia where ASFV is spreading?
https://hhv6.jottit.com/35._pigs_and_kaposi%27s_sarcoma_in_sardinia
The world's highest incidence of Kaposi's sarcoma occurs in Sardinia (Reference) Is it possible that it is due to the fact that African Swine Fever Virus is endemic on the island? (Reference) One study suggests that the incidence of K.S. in northern Sardinia is highest in a countryside area where people have contact with animals. (Reference) Given the high prevalence of HHV-8,--the so-called K.S. herpes virus--in Sardinia (Reference) is it at all possible that HHV-8 may have been misclassified and actually is a human-adapted form of African Swine Fever Virus? (ASFV has been at least visually mistaken for another herpes virus, CMV, in the past.)
A number of experiments could be conducted to explore this hypothesis. In addition to a direct comparison of ASFV and HHV-8, pigs with African Swine Fever Virus could be tested for sequences of HHV-8. People with Kaposi's sarcoma could be tested for sequences of African Swine Fever, including new Asfaviridae sequences recently discovered. (Reference)
A comparison of the K.S. lesions in humans and ASFV lesions in pigs might be in order.Given that African Swine Fever is currently spreading in Russia and is now threatening Europe and China, (Reference) it would be useful to know whether people who are exposed to pigs with ASFV are at increased risk for HHV-8, Kaposi's sarcoma and the other pathologies associated with HHV-8. A study in sub-Saharan Africa where ASFV is endemic and HHV-8 is also endemic (Reference) might be useful. And areas of Russia where ASFV is spreading could be monitored closely for any signs of an increase of K.S. or HHV-8 infection and HHV-8 related pathologies.HHV-8 is an emerging health problem. HHV-8-associated K.S. is a significant problem in AIDS patients. It may also be the key to Chronic Fatigue Syndrome. HHV-8 has been found in the cerebrospinal fluid of 50% of Chronic Fatigue Syndrome patients. (Reference) HHV-8 has been linked to type 2 diabetes. (Reference) HHV-8 has been detected in B-cells in Castleman's disease and primary effusion lymphoma. (Reference).
If HHV-8 is a form of ASFV, it is possible that pigs might constitute a useful animal model for the study of possible treatments for K.S. and other pathologies associated with HHV-8. And if there is any relationship between ASFV and HHV-8, people may have to be warned to take special precautions around pigs in areas where there are ASFV outbreaks. And countries where undercooked pork is consumed (like Ukraine where salo is a staple) may need to alert the public to cook all pork products thoroughly during ASFV epidemics.
A number of years ago, Neenyah Ostrom reported in the New
York Native on the lesions of CFS patients which seem
to resemble Kaposi's Sarcoma (KS). The current
thinking is that a virus called HHV-8 is the cause of
KS. (Although HHV-6 has recently also been implicated
once again.) If KS is a problem in CFS (and we
suspect it is) then one should be able to find HHV-8 and
HHV-6 in CFS patients. Apparently, in this small
study, one can. Below is a rather explosive abstract:
York Native on the lesions of CFS patients which seem
to resemble Kaposi's Sarcoma (KS). The current
thinking is that a virus called HHV-8 is the cause of
KS. (Although HHV-6 has recently also been implicated
once again.) If KS is a problem in CFS (and we
suspect it is) then one should be able to find HHV-8 and
HHV-6 in CFS patients. Apparently, in this small
study, one can. Below is a rather explosive abstract:
Prevalence in the cerebrospinal fluid of the following
infectious agents in a cohort of 12 CFS subjects:
human herpes virus-6 and 8; chlamydia species;
mycoplasma species; EBV; CMV; and Coxsackievirus.
Levine, S.
Journal of Chronic Fatigue Syndrome, 2001, 9, 1/2,
41-51.
infectious agents in a cohort of 12 CFS subjects:
human herpes virus-6 and 8; chlamydia species;
mycoplasma species; EBV; CMV; and Coxsackievirus.
Levine, S.
Journal of Chronic Fatigue Syndrome, 2001, 9, 1/2,
41-51.
Abstract:
Over the last decade a wide variety of infectious
agents have been associated with the CFS as potential
etiologies for this disorder. Many of these agents are
neurotrophic and have been linked previously to other
diseases involving the central nervous system (CNS).
Human herpes virus-6 (HHV-6), especially the B
variant, has been found in autopsy specimens of
patients who suffered from MS. Because patients with
CFS manifest a wide range of symptoms involving the
CNS as shown by abnormalities on brain MRIs, SPECT
scans of the brain and results of tilt table testing
we sought to determine the prevalence of HHV-6, HHV-8,
Epstein-Barr Virus (EBV), cytomegalovirus (CMV),
mycoplasma species, chlamydia species, and Coxsackie
virus in the spinal fluid of a group of 12 patients
with CFS (CDC criteria '94).
Over the last decade a wide variety of infectious
agents have been associated with the CFS as potential
etiologies for this disorder. Many of these agents are
neurotrophic and have been linked previously to other
diseases involving the central nervous system (CNS).
Human herpes virus-6 (HHV-6), especially the B
variant, has been found in autopsy specimens of
patients who suffered from MS. Because patients with
CFS manifest a wide range of symptoms involving the
CNS as shown by abnormalities on brain MRIs, SPECT
scans of the brain and results of tilt table testing
we sought to determine the prevalence of HHV-6, HHV-8,
Epstein-Barr Virus (EBV), cytomegalovirus (CMV),
mycoplasma species, chlamydia species, and Coxsackie
virus in the spinal fluid of a group of 12 patients
with CFS (CDC criteria '94).
We found evidence of HHV-6, HHV-8, chlamydia species,
CMV and Coxsackie virus in 6/12 samples. Plasma tests
were negative. It was surprising to obtain such a
relatively high yield of infectious agents in cell
free specimens of spinal fluid that had not been
centrifuged. Future research in spinal fluid analysis,
in addition to testing tissue samples by polymerase
chain reaction (PCR) and other direct viral isolation
techniques will be important in characterizing
subpopulations of CFS patients, especially those with
involvement of the CNS.
CMV and Coxsackie virus in 6/12 samples. Plasma tests
were negative. It was surprising to obtain such a
relatively high yield of infectious agents in cell
free specimens of spinal fluid that had not been
centrifuged. Future research in spinal fluid analysis,
in addition to testing tissue samples by polymerase
chain reaction (PCR) and other direct viral isolation
techniques will be important in characterizing
subpopulations of CFS patients, especially those with
involvement of the CNS.
The low rate of isolation of HHV-6 may be related to
the lack of gross neurological findings in the
patients at the time of testing.
the lack of gross neurological findings in the
patients at the time of testing.
An overview of KS:http://www.thebody.com/nmai/ks.html
Except for those who have made a lifelong commitment
to denial, finding the so-called "KS virus" (HHV-8)
and the "supporting KS virus" (HHV-6) in CFIDS patients
should help settle the question of the overlapping
nature of the AIDS and CFIDS epidemics.
to denial, finding the so-called "KS virus" (HHV-8)
and the "supporting KS virus" (HHV-6) in CFIDS patients
should help settle the question of the overlapping
nature of the AIDS and CFIDS epidemics.
More info on KS here.
Given that HHV-8 is part of the HHV-6 and HHV-7 family, this may be very important:
Journal of General virology (1995), 76, 147-152
Click here for more information on this issue.
Given that HHV-8 is part of the HHV-6 and HHV-7 family, this may be very important:
"The 19R Protein of HHV-6 has significant amino acid sequence homology . . . to a protein encoded by African Swine Fever Virus."
--Glenda L. Lawrence, John Nicholas and Bart G. BarrellJournal of General virology (1995), 76, 147-152
Click here for more information on this issue.
Saturday, April 11, 2015
Viruses Every Cat, Dog, Human and Pig Should be Tested for in Russia and Europe
As a sentinel system for an African Swine Fever epidemic in pets, humans and pigs, Russian and European scientists should test cats, dogs, humans and pigs for these viruses. They may all be related to each other and may interact with each other in causing immune damage and other problems.
1. HHV-6
2. HHV-7
3. HHV-8
4. Porcine Lymphotropic Herpesvirus 1
5. Porcine Lymphotropic Herpesvirus 2
6. African Swine Fever Virus
1. HHV-6
2. HHV-7
3. HHV-8
4. Porcine Lymphotropic Herpesvirus 1
5. Porcine Lymphotropic Herpesvirus 2
6. African Swine Fever Virus
Friday, April 10, 2015
Is it possible that human infections of African Swine Fever Virus will be the first sign that the disease has hit Western Europe?
"Since antibodies for the African Swine virus have been detected in
humans, the possibility of human infection with the African Swine virus
exists and may thus far have escaped any systematic screening. Thus, any
preventive and therapeutic approach to African Swine fever can have
far-reaching implications to control immune deficiency conditions in
humans."
Read more: http://www.faqs.org/patents/app/20080207875#ixzz3Wwwc8X9T
"While the African Swine virus has been primarily detected in pigs and certain other animals, antibodies against the African Swine virus have also been found in humans (5). The fact that there was no description of any finding of the African Swine virus in humans may thus be attributable to oversight or a lack of understanding for the significance of African Swine fever virus for the pathogenicity of immune deficiencies in humans."
Read more: http://www.faqs.org/patents/app/20080207875#ixzz3Wwwc8X9T
"While the African Swine virus has been primarily detected in pigs and certain other animals, antibodies against the African Swine virus have also been found in humans (5). The fact that there was no description of any finding of the African Swine virus in humans may thus be attributable to oversight or a lack of understanding for the significance of African Swine fever virus for the pathogenicity of immune deficiencies in humans."
So far, in this article, I have listed a total of 20 factors that ASFV and HHV-6A have in common. Ten are common areas of symptomology and 10 are particular characteristics of the virus. What is just as significant is the absence of clinical evidence that shows a difference between the two viruses. The only real test to settle the issue is for researchers to attempt to infect a pig with blood from an AIDS patients with Lymphadenopathy and see if it brings on African Swine Fever. If the real virus that causes AIDS came from pigs, then infecting them with the AIDS virus (HHV-6A) should cause them to develop ASFV. If it does, it will be case closed. Are there any researchers reading this article that are willing to do an in-vivo test on live swine? America's Biggest Cover-up A book came out late in Nov., 1993, titled America's Biggest Cover-up, by Neenyah Ostrom. It alleges that HHV-6 (A) may be the primary causative factor in both AIDS and Chronic Fatigue Syndrome. There are two strains of HHV-6, variants A and B. HHV-6 (Variant B) is a common herpes virus and is not a life threatening infection. Ostrom, a prolific researcher and writer, has written articles for The New York Native on HHV-6 and other AIDS related topics for several years (1). Ostrom says the variant A strain of HHV-6 is the culprit in both CFS and in AIDS. Ostrom has interviewed most of the major researchers in the field, as well as countless patients and government officials. She has found many similarities between Chronic Fatigue Syndrome and AIDS and believes that they are part of the same epidemic. She argues that until their connection is admitted by top government researchers, there is little hope that real progress will be made in stopping these two expressions of the same syndrome. Ostrom also says in her book that HHV-6A is really the African Swine Fever virus (ASF). Ostrom reports that CFS patients have lost much of their B cell and natural killer (NK) cell protection and both groups are susceptible to night sweats and many forms of cancer, lymphomas and TB. The book is available at your local bookstore.
Thursday, April 9, 2015
Wednesday, April 8, 2015
Are Russian Scientists Aware that American Scientists May Have Renamed African Swine Fever, Calling it HHV-6?
John Beldekas on African Swine Fever Virus and Gallo's peculiar "discovery" of HHV-6
In August, 1986, John Beldekas was invited to go to the NCI and present his findings on the link between ASFV [African Swine Fever virus] and AIDS, which he did. Beldekas gave samples of all his lab work to Gallo. Later, the government asked Beldekas to turn over all his reagents and lab work to the government, which he did. Beldekas had found ASFV presence in nine of 21 AIDS patients using two standard procedures. At the meeting, Gallo was reported saying: “we know it is not ASFV.” How could Gallo know this as he hadn’t done any of his own tests to look for ASFV?
Two months later, Gallo published an article in Science (Oct 31, 1986) that he discovered a new possible co-factor in AIDS, a virus he called Human B Cell Lymphotropic Virus which he named HBLV. Like ASFV, HBLV infected B cells and also lived in macrophages. Did Gallo steal Beldekas’s ASF virus he found in AIDS patients and rename it HBLV? Later on, when Gallo found that HBLV could also infect other immune cells, he changed the name of HBLV to HHV-6. Eventually, Gallo identified his HBLV as the variant A strain of HHV-6 and called it a human herpesvirus.
--Mark Konlee
http://www.keephopealive.org/report10.html
In August, 1986, John Beldekas was invited to go to the NCI and present his findings on the link between ASFV [African Swine Fever virus] and AIDS, which he did. Beldekas gave samples of all his lab work to Gallo. Later, the government asked Beldekas to turn over all his reagents and lab work to the government, which he did. Beldekas had found ASFV presence in nine of 21 AIDS patients using two standard procedures. At the meeting, Gallo was reported saying: “we know it is not ASFV.” How could Gallo know this as he hadn’t done any of his own tests to look for ASFV?
Two months later, Gallo published an article in Science (Oct 31, 1986) that he discovered a new possible co-factor in AIDS, a virus he called Human B Cell Lymphotropic Virus which he named HBLV. Like ASFV, HBLV infected B cells and also lived in macrophages. Did Gallo steal Beldekas’s ASF virus he found in AIDS patients and rename it HBLV? Later on, when Gallo found that HBLV could also infect other immune cells, he changed the name of HBLV to HHV-6. Eventually, Gallo identified his HBLV as the variant A strain of HHV-6 and called it a human herpesvirus.
--Mark Konlee
http://www.keephopealive.org/report10.html
Subscribe to:
Posts (Atom)
Our books on Amazon
